I am sorry you are yet another whose physician knows little and neither does the nurse.
If you email email@example.com and ask for a cop of Dr Toft's Pulse online article. Read question 6 and make a new appointment and insist on some 20mcg of T3 being added to your reduced 125mcg of levo (I'm glad you didn't reduce from 150mcg).
Also they should have tested your Free T3 as that's the most efficient to test. Your T4 is too low and I doubt you are converting sufficient into T3. T3 as you most probably know is the Active hormone and we cannot function normally with too little.
Your pulse is far too low and it's very obvious you're not on sufficient. They are also going by the TSH only. Give them both a copy of the following so they can read and learn as we have to do,unfortunately,if we're to recover some semblance of good health.
The way doctors used to diagnose before these blood tests were introduced which should really be used for information rather than a diagnosis was to take our temp before rising and pulse plus our clinical symptoms.
As your pulse is so low and your heart has to work harder
Excerpt from above:
August 17, 2004
Because her heart’s response to the stress hormones is blunted, the heart pumps too little blood into her general circulation. The resulting less-than-adequate flow of blood to her exercising muscles leaves them deficient of oxygen. And the oxygen deficit renders the muscles unable of accommodating her desire to exercise hard enough to lose weight.
I want to emphasize that the TSH test has nothing whatever to do with guarding a patient’s cardiac safety. To infer that a patient has cardiac over-stimulation because the TSH level is low is scientifically unjustified and logically unsound. It is ludicrous for a doctor to make this inference when he can easily and directly monitor how the patient’s heart is responding.
Let me emphasize another important point: Some conventional endocrinologists have grossly exaggerated the cardiac risks of TSH-suppressive doses of thyroid hormone. When compared to replacement doses of thyroid hormone, TSH-suppressive doses are not associated with an increased incidence of ischemic heart disease. In fact, TSH-suppressive doses of thyroid hormone protect the heart. TSH-suppressive dosages lower the levels of blood fats more than replacement doses do. And higher-end doses of thyroid hormone can halt the progression of coronary artery disease. In patients who don’t have coronary artery disease, myocardial ischemia and/or infarction are rare even in those who are thyrotoxic.