Just testing TSH on it's own is not enough, we also need to test FT4 because one can have a normal TSH but low FT4 which would suggest Central Hypothyroidism (which is where the problem lies with the pituitary rather than the thyroid ).
Please add reference ranges for all your results so that we can interpret them, ranges vary from lab to lab .
Iron panel looks low, was saturation percentage also done?
Vit D is likely to be low, is the unit of measurement nmol/L or ng/ml?
I've just started wellwomans vitamins to see if helps tiredness. Doctor said all results normal. On no medication except sertraline slowly cutting down.
My symptoms are tiredness dry eyes hair loss brain fog.
Serum iron is bottom of range and ferritin at 6.25%. TIBC measures the transferrin proteins available for binding free iron in the body and is way under range.Â
SlowDragon refers to the usual lab presentation where elevated TIBC may indicate iron deficiency anaemia (too many proteins available for iron to bind to) and low TIBC may indicate iron overload (too few proteins available for iron to bind to). However, when we are long term ill iron mechanisms can go askew and usual patterns don’t always follow through. Low iron and low TIBC could be due to conditions such as haemolytic anaemia where RBC are destroyed too quickly or protein levels become low through inflammation.Â
Again a usual presentation of inflammation would be for ferritin to raise to secure iron from feeding pathogens but if you don’t have any iron or proteins are low due to other conditions, then ferritin isn’t going to raise. There are many forum members with high inflammation evidenced by swellings, aches & pains, elevated auto antibodies, etc but low ferritin.
There is also thought when iron behaviours are left long term as in the case of too little, then the amount of transferrin protein carriers will naturally reduce to correlate with the low level of iron and in which case you would end up with low iron and low transferrin.
Haemolytic anaemia presents as primary, secondary and autoimmune with the cold temps common in hypothyroidism impairing the production of RBC’s (as explained by Dr P in his book).Â
Definitely no haemochromotosis as you are deficient in iron. Have you had a FBC to assess RBC health and haemoglobin levels?
Also a WellWomen multivitamin will not address deficiencies. As you are low in iron and Vit D it would be prudent to assume malabsorption issues and a need for further nutrient testing/monitoring.
Low iron and low TIBC could be due to conditions such as haemolytic anaemia where RBC are destroyed too quickly or protein levels become low through inflammation.Â
Another possible cause for low iron and low TIBC in the same person is internal or GI bleeding. My iron and TIBC ended up below range thanks to a bleeding polyp.
My diet is mixture of things have meat twice a week. Tend to skip breakfast 🙈 sometimes bowels go from one extreme to another but not too bad.Do get bloating.
My main symptoms are tiredness, dry eyes, brain fog. It's so hard isn't it when doctor says all ok. But hard to keep going back x
Do you know roughly how much private blood tests are for thyroid?Be so great to get to the bottom of it so hard to know if vitamin D causing tiredness or iron or autoimmune diesese 😬 so hard keep ringing doctor especially when says all normal x
Vitamin D deficiency is frequent in Hashimoto's thyroiditis and treatment of patients with this condition with Vitamin D may slow down the course of development of hypothyroidism and also decrease cardiovascular risks in these patients. Vitamin D measurement and replacement may be critical in these patients.
Vitamin D insufficiency was associated with AITD and HT, especially overt hypothyroidism. Low serum vitamin D levels were independently associated with high serum TSH levels.
The thyroid hormone status would play a role in the maintenance of vitamin D sufficiency, and its immunomodulatory role would influence the presence of autoimmune thyroid disease. The positive correlation between free T4 and vitamin D concentrations suggests that adequate levothyroxine replacement in HT would be an essential factor in maintaining vitamin D at sufficient levels.
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