If the body needs cortisone to convert T4 to T3, then why does administering hydrocortisone during thyroid storm inhibit peripheral conversion of T4 to T3?
Page 678 indicates that cortisone is used to treat thyrotoxicosis, for this reason.
My partner's cortisone level is very low but as soon as he took his first dose of hydrocortisone he ended up in the hospital with all the symptoms of thyrotoxicosis. He still has all the symptoms, of what he calls being "hyper" I assume that means thyrotoxicosis - but, of course, the hospital did nothing but release him as soon as it was apparent he didn't have a heart attack. I can't understand that. That's NOT how things are done in America (where I am). He would have been tested, treated, and remained there until well. I can't understand how any of this is happening. It's like a 3rd world situation.
Now, I have no idea if I should tell him to take the hydrocortisone to prevent an adrenal crisis, which could kill him. Or, if I should tell him not to take it because he got instantly ill last time and he's still suffering the same symptoms. Also, he's petrified to take it. So, I have no idea what to do. I'm trying to understand if hydrocortisone is safe or not. If he's been over medicated with levo, then is taking it going to cause a jump in T3? If so, then why is it used to treat thyrotoxicosis? Where can I get these answers?