This will only make sense if you’ve read my previous post
healthunlocked.com/rlsuk/po....
There was a question in this post and I promised the writer of the best answer a chocolate muffin.
It’s my opinion that the best information was provided by Ironbrain, so the muffin is for you.
Sorry, I can’t literally send one to you. I have no idea what condition it might be in by the time it reached you.
My simplistic interpretation of what happens to iron when it get’s into the brain in relation to RLS and nothing else is this. (See ironbrains' links below).
Iron is taken into brain cells (neurons) and some of these will be involved with RLS. In these cells some iron is taken up by structures called mitochondria and some iron remains in the liquid that’s found in the cell (cytosol).
The iron in the mitochondria may perform particular functions, whereas the iron outside (cytosol) performs different functions.
The iron outside plays a part in normal dopamine and adenosine function, without which RLS can occur.
Apparently, it has been found that there is both an excess of mitochondria and an excess of iron in the mitochondria in RLS and hence, on balance, insufficient iron outside thus compromising dopamine and adenosine functions.
academic.oup.com/jnen/artic...
Unfortunately, other studies find a lack of mitochondrial iron.
movementdisorders.onlinelib....
MY story has ended, but for me the end of the story of this elusive condition has again eluded me
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FINALLY found a scientific study that supports my theory about Brain Iron and Serum Iron. Finally!!!
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