You all know that I only have a high school degree, but I did have a pretty successful career in Process and Training for a huge international telecommunications company.
I just mention this because one of the things I brought to the table at work was a willingness to state the obvious. A lot of people avoid stating the obvious. I don't know why that is.
So... on our Sunday Zoom call (you all should join us!) we had a new member telling us about how his Parkinson's started. I think it started with a stress tremor in one hand. Then it progressed to other parts of the body and other symptoms.
So... this is my obvious observation: If PD symptoms are caused by a lack of dopamine in the body, caused by dopamine creating cells in the substantia nigra dying or shutting down, then why do symptoms start in one place?
I think the symptoms start in one place because there is a lot more happening that the substantia nigra not creating enough Dopamine. This is probably why Dopamine replacement does not alleviate all symptoms (because the symptoms are not all due to lack of Dopamine).
If you Google "structural brain changes Parkinson's" you will find a lot of not pleasant information on changes to the brain with PD. If we are biohacking, this stuff is probably worth thinking about. Can we address these specifics?
I don't have the research in front of me, but I have read about how PwP brain frequencies tend to homogenize at around 13 HZ. This is why I use brainwave entrainment every day at 40 HZ.
I also have a file if anybody wants is (I made it myself!): Monaural Beats Files: There is only one. This is my favorite file now. It is 250 HZ pulsed at 40 HZ (Gamma). I chose 250 HZ because I think that is the frequency of the Tass gloves. Here is the file: 250hz pulsed at 40hz Isochronic.mp3 drive.google.com/file/d/14X...
But this post is not just about brainwave entrainment. It's about thinking about more than lack of dopamine as we try to stop progression.
Okay, I will shut up now.
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neurons have long thread-like structures that connect them electronically to other neurons. These structures are called axons and dendrites. Axons transmit information from their cell to the dendrite of another cell across a gap called a synapse. Dopamine is a neurotransmitter. It is passed like a message across the synapse. When neurons atrophy these dendrites shorten, axons weaken, withering away, and less dopamine is produced so communication between neurons is weakened or disrupted. Just like in telecommunications, bad connections cause messages to fail. Dopminergic neurons have dendrites that stretch all throughout the brain. Depending on the network involved, you may experience deficits in the corresponding areas, such as upper right extremity, lower left extremity, etc. This is an oversimplified explanation but I think you get the picture. .
brainwave entrainment, as you call it, can help with neuroplasticity, which is the strengthening and regrowing of brain neurons. It can help, potentially, and at the very least slow down the progression of the damage.
Typically, it takes years of atrophy to get to the point of manifesting symptoms. The Substantia Nigra is usually 70% to 80% obliterated by the time symptoms start. Neuroplasticity can be like running against the wind and uphill. If you can identify the cause of your particular case, and stop whatever it is, then maybe you can turn it around 180 degrees. Even stem cells have limited results because, although they can function as dopaminergic cells, whatever killed the original cells are going to kill the new cells as well,, and, they don’t really get a chance to develop new versions of the original connections that withered away.
I especially appreciate this: "When neurons atrophy these dendrites shorten, axons weaken, withering away, and less dopamine is produced so communication between neurons is weakened or disrupted. Just like in telecommunications, bad connections cause messages to fail. Dopminergic neurons have dendrites that stretch all throughout the brain. Depending on the network involved, you may experience deficits in the corresponding areas, such as upper right extremity, lower left extremity, etc." and the corresponding "they don’t really get a chance to develop new versions of the original connections that withered away".
And I might add… If PD symptoms are caused by a lack of dopamine in the body, caused by dopamine creating cells in the substantia nigra dying or shutting down, then why am I able to (with considerable effort) calm my mind and body to the point where my tremors stop? And then once my mind kicks in again, they start up immediately. There’s gotta be more to it than just a lack of dopamine.
My HWP uses the red light that is 40hz alternating with another light. I assume this is different. So if we want to get started with this treatment is there a dummy version on what to buy and how it works?
There is nothing to buy if you want to try this. I posted the link to the audio file above and you can listen to it on headphones or on a speaker or really any way.
This is not any type of approved treatment. Just me taking a shot at something that is no cost and, based on some research, might have a benefit. Please check out these two posts:
I started with binaural beats but I really could not hear them. Then I saw the MIT study that used monaural beats with mice ad some other things and I started thinking monaural was better and simpler.
Yes, reading the responses I realized I had that backwards. Especially when I know darn well that the purpose of Carbidopa is to get the Levadopa past the BBB and into the brain.
Now maybe people will believe me when I say I am not a smart man.
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Gamma brain wave entrainment using sound and light at 40 Hz
Men Get PD Significantly More Than Women And The Relationship Of Melatonin In These Two Populations
Frequency difference between tremors and dyskinesia
