This is probably one for the scientists: haptocorrin seems to be 1st phase binder to B12 and comes from the salivary glands and then on to the stomach, where haptocorrin's main task seems to be to protect B12 from hydrochloric acid produced by parietal cells. Am I right? If this is so, can a saliva gland infection and blockage and deformation of saliva ducts eventually become a B12 problem?
As you can see, still trying to find a reason, and perhaps solution. Don't want to bark up any more trees particularly with Dr, unless there could be something here?