In a recent study, published in The Journal of Neuroscience, researchers adopted a novel approach to dyskinesia by treating it as a “bad motor memory.”
They discovered that inhibiting a protein called Activin A could stop dyskinesia symptoms and essentially erase the brain’s “bad memory” response to certain Parkinson’s therapies.
Rather than seeking an entirely new treatment, researchers aimed to find a way to prevent dyskinesia from developing in the first place.
Please, provide some links to "levodopa doesn't cause dyskinesia". And, if not caused by, who cares, if it can be stopped.
CuriousMe12 Maybe the notion that C/L doesn't cause dyskinesia is the only non-(pure science) posted in this post?
I question why the researches call it memory – Maybe, a programmed memory, (a subroutine the brain runs to react to stimuli (Levodopa).
So what is the role of this molecule and does it play role in other brain and neurological functions, or just the one? (Meaning: am I going to lose mentation as the cost for the benefit of reducing dyskinesia?) and who does it benefit, and who does it not benefit?
Apologies. I shot from the hip.🤠
Long-term treatment with transcranial pulsed electromagnetic fields improves movement speed and elevates cerebrospinal erythropoietin 2021
Long‐Term Safety and Clinical Effects of Nilotinib in Parkinson's Disease (Nov 20, 2020) 
Interesting Gender Research Findings RE: Levodopa Absorption RatesCDP choline and dyskinesia 
Has anybody found a way to reduce dyskinesia without Amantadine?
