This "chicken and egg" question occurred to me while reading an interesting article [1] concerning Amyotrophic Lateral Sclerosis (ALS).
I also saw parallels with some aspects of PD research.
"These proteins end up collapsing the cellular clearance systems, which finally leads to the death of the motor neurons."
"We must find ways to reduce the production of ribosomes so that waste decreases, while still keeping a sufficient number to guarantee the correct functioning of the cells." (c.f. the posiphen approach to reducing alpha synuclein)
[1] MedicalXpress article: Accumulation of 'junk proteins' identified as one cause of aging and possible source of ALS.
Not enough dopamine for Dystonia vs. too much dopamine creating fatigue and daytime sleepiness.
Can we give CuATSM a try without waiting for the Australian trial completion (5 years from now)
phosphatidylserine Neuroprotective? Works with DHA , reduces insomnia 
my first ever poem…..
