mdpi.com/2076-3921/10/9/148...
chartist please help decipher
Conclusions
The physiological and pathological functions of biomolecular condensates in neurodegenerative disorders are shaped by powerful, complex, interdependent relationships between membraneless organelles, membranes/lipid rafts, ATP, RNA, and most of all, stress and its timely resolution. Melatonin’s intimate association with each of these decisive influencers may position the potent, ancient antioxidant as an important mediator of the phase separation of condensates in health and disease via principal ATP-dependent mechanisms including post-translational modifications and RNA m6A modifications (Figure 1). This novel theoretical review is presented with the intention to spur further research interest and exploration in the full, multi-faceted potential of melatonin in the regulation of biomolecular condensates that could provide solutions and answers to existing and future challenges and questions in this exciting and promising field of study.
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