Left branch bundle block.

[Whitenightingale]

Has anyone here found they have a Left branch bundle block after they were diagnosed and on thyroxine? It is to do with the electricity in the ❤️ heart.

[Polina44]

Hi,

I've recently been told I have Right bundle branch block and not to worry about it. Found in ECG after chest pain. I take thyroxine, liothyronine and hydroxychloroquine (for SLE).

L 🙃

[Whitenightingale in reply to Polina44]

I had chest pain and irregular heartbeat a year on 50 mg thyroxine. I think I was on a too low dose and this was the result. My heart was struggling, or that is how I was feeling. Recently I was told yo reduce from 125 to 100 and then I got it all back it was making me cough when the beats returned after missing one. At night time it felt worse. Now it seems to have righted itself but the ECG showed differently to a previous one and the doctor has said it's detiorated since the last ECG. He is now going to send to the Cardio Dept at the hospital to get an opinion. So will be interesting to see what they say as last time .I saw them they said I would be fine to my eighties!

[shaws]

I haven't heard of that but I do know that if hypothyroid and not on sufficient thyroid hormones our heart can be affected. Most doctors, I think, don't know this either.

Excerpt:-

I want to emphasize that the TSH test has nothing whatever to do with guarding a patient’s cardiac safety. To infer that a patient has cardiac over-stimulation because the TSH level is low is scientifically unjustified and logically unsound. It is ludicrous for a doctor to make this inference when he can easily and directly monitor how the patient’s heart is responding.

Let me emphasize another important point: Some conventional endocrinologists have grossly exaggerated the cardiac risks of TSH-suppressive doses of thyroid hormone. When compared to replacement doses of thyroid hormone, TSH-suppressive doses are not associated with an increased incidence of ischemic heart disease. In fact, TSH-suppressive doses of thyroid hormone protect the heart. TSH-suppressive dosages lower the levels of blood fats more than replacement doses do. And higher-end doses of thyroid hormone can halt the progression of coronary artery disease. In patients who don’t have coronary artery disease, myocardial ischemia and/or infarction are rare even in those who are thyrotoxic.

Moreover, restricting many patients to replacement doses predisposes them to cardiovascular disease and premature death. With these patients, erring on the side of safety means one thing—allowing them to use higher-end rather than lower-end doses of thyroid hormone.

In most patients, then, TSH-suppressive doses of thyroid hormone don’t harm the heart. This justifies Dr. L.E. Shapiro writing in 1997, "In the absence of symptoms of thyrotoxicosis, patients treated with TSH-suppressive doses of L-T4 may be followed clinically without specific cardiac laboratory studies."[2]

web.archive.org/web/2010103...

[Naomi8]

I was diagnosed with this a few years ago & recently offered ablation to stop the supraventricular tachycardia episodes,whenever I want.I was told it was an electrical fault caused by a birth defect.

Since going on to T3-only over a year ago & giving up thyroxine,I have not had any episodes.

That was until I tried NDT.I tried 2 trials of 5 weeks each,but due to a return of tachycardia & ectopics,I have gone back to T3-only.

[Whitenightingale in reply to Naomi8]

Yes it is an electrical issue. I do t think I had it until I started on low doses of thyroxine I think I was kept on a too low dose and it damaged the electrical system.