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Parkinson's Movement
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Haemochromatosis and Parkinsons Disease

Haemochromatosis and Parkinsons Disease

There is a family history of Haemochromatosis.....my Mother, my Sister, a cousin and I am a Carrier. I also have Parkinsons Disease.

I would like to find more information on the relationship. Only today did l find a Link to the above.

The Carrier State was diagnosed long before PD. I have not thought to mention my Carrier State to my Neurologist.....should I ?


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There is a relationship between Primary haemochromatosis in Parkinson's disease because the condition causes a systemic build up of iron within the body. And iron is a known risk factor for Parkinson's disease:


I have the type 2 anemia trait (I do not require blood transfusions) and read Dr. Thomas Emery's book "Facts and Fallacies, Iron and Your Health" and though he does not mention haemochromatosis in relationship to PD he certainly talks about Primary and Secondary haemochromatosis:


In Emery's book he noted that Southern French wine has the highest content of iron of all wines and you should avoid it. He notes there are 2 substances which faciliate the absorbtion of iron: vitamin c and alcohol - hence the high alcohol content of 'Geritol'.

If I had both conditions I would take a high potency green tea extract with high EGCG content and purity. The following article notes the many different iron chelators available and it does get around to EGCG:


Also, EGCG is best absorbed with 200 mg vitamin C, 1,000 mg salmon omega 3 on an empty stomach with no calcium, milk or hard water, The following article tells how EGCG is best absorbed by the body:


I recommend the following supplement for its purity and price. Disregard the bottle because they recommend taking the capsule with food------ does not anyone do research anymore?



Thank you Silvestrov.

I need now to take it further with my professional team....


Interesting discussion

Here is another piece for the puzzle

Iron Accumulation in Parkinson’s Disease: Cause or Effect?

Is iron elevation contributing to neuronal death in Parkinson’s disease, or is it simply a feature of dying neurons? To address this important question, this paper will review three separate lines of evidence: (1) in vivo iron-imaging technology, (2) rare genetic disorders of iron metabolism, and (3) animal models.




I shall print this out and try to understand it.

I am not a scientist but shall enjoy discussing it with other PD friends and see what they come up with. Also thrash it out with my wonderful Neurologist who expects me to turn up with something I have found on the net.

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