I have stage 4 ckd. Last GFR was 29 so actually right on the border of 3/4. I have the flu and I have noticed ever since being sick my urine is very bubbly. This happened when I had Covid last year but then it went back to normal. I am drinking my water but cannot eat so it has been affecting my sugar level. the only thing I can eat is some white toast and an egg. Just thinking about food makes me nauseous. I have only taken a few Tylenol pills for the fever. Has anyone else experienced this when sick? Thank you
Yankees28
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Yankees28
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There are other reasons for bubbly urine, not just protein. Could be medications, dehydration, and speed of urine stream. Try not to worry, but keep an eye on it. If the same thing happened to you with COVID, it could just go away when you're feeling better. Usually when we have the flu (and especially fever), we get dehydrated. Take care, and hope you're feeling better soon!
The rule of thumb is that if you're properly hydrated, all the bubbles will burst before 5 minutes. If they're lingering after that, it may be due to protein leakage.
Have you gone to your doctor asking for a blood and a urine test to verify how your kidneys are doing? With your numbers close monitoring is important.
Clinical Significance of Subjective Foamy UrineKyu Keun Kang, Jung Ran Choi, Ji Young Song,Sung Wan Han, So Hyun Park, Woong Sun Yoo,Hwe Won Kim, Dongyoung Lee,Kyoung Hyoub Moon, Myung Hee Lee,1 and Beom KimAuthor
information Article notes Copyright and License information DisclaimerGo to:Abstract
Foamy urine is widely regarded as a sign of proteinuria. However, there is no objective definition of foamy urine and there are no reports on the proportion of involved patients who have overt proteinuria or microalbuminuria. We performed this study to investigate this proportion and to identify possible risk factors for these two conditions. We reviewed all new outpatients from 1 November 2011 to 30 April 2012 and identified patients complaining of foamy urine. Their demographic data and medical records were examined. In particular, we tabulated the patients' spot urinary protein to creatinine ratio, spot urinary microalbumin to creatinine ratio (ACR), blood urea nitrogen (BUN), and serum levels of creatinine (Cr), uric acid, calcium, phosphate, and glucose. In addition, we calculated estimated glomerular filtration rates (eGFRs) by using the CKD-EPI equation. We also performed risk factor analysis with the Chi-squared test and by logistic regression. Seventy-two patients (6.3% of total new outpatients) complained of foamy urine; of these, there were 59 males with a median age of 65.5 years (range, 36-87 years). Of the 72 patients, 16 (22.2%) had overt proteinuria. We found that diabetes, poor renal function (high Cr, BUN, low eGFR), increased serum phosphate, and increased serum glucose were associated with overt proteinuria. Multiple logistic regression analysis showed that serum Cr and serum phosphate were associated with overt proteinuria. The ACR was available for 38 patients, and in this subgroup, 12 (31.6%) showed microalbuminuria or overt proteinuria. In this subgroup, a high serum Cr was the only statistically significant risk factor. Among patients who complained of foamy urine, approximately 20% had overt proteinuria, and increased serum Cr and phosphate were statistically significant risk factors.
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