I've been a Diabetic for the last 15 years. Presently, I am using Allopathy medicines (including Insulin). Can my Diabetes be cured totally with Ayurvedic medicines? Thank you.
Mukkamala
I've been a Diabetic for the last 15 years. Presently, I am using Allopathy medicines (including Insulin). Can my Diabetes be cured totally with Ayurvedic medicines? Thank you.
Mukkamala
This can be answered only by ayurvedic doctor
Just Ayurveda or homeopathy or allopathy or any other pathy does not cure until and unless life style changes are incorporated .
Agree with coffeeday!
Diabetes is not a disease. It is a lifestyle disorder. If you correct your lifestyle , you can reverse your type 2 diabetes .
Since it is not a disease no pathy can cure it. Rather , all modern medicines in combination will add to the complexity of the disease.
If you are taking Allopathy, has it cured ???
If some so called ayurvedic medicines can cure , then people will definitely try and finally everybody would get cured ! In India , the govt- Ayush has also approved two ayurvedic drugs for type 2 diabetes namely BGR 34 and IME 9 . Please see the review in Amazon.in . The result is not consistent. And the long term side effects of these so called Natural herbal drug is not yet established.
At least for the Allopathy, the possible side effects are listed by the manufacturer and by USFDA. So you are aware.
I fully agree with the reason given by Mr namah.So by changing life style and food and of course little medicine we can reverse it.
Yes I am also agree with namah changing life style for revrse type 2 diabetic I.e eating two times a day ,my hba1c came down from 8.9 to 6.5 by two time eating only eat whatever may be but only two time lunch & dinner but no sugar items
That is great! Congrats !
For our learning , can you please tell me:
1.How many kcal / day is your total Intake with two meals ?
2. What is your current BMI?
Thanks
Best regards
Daily intakes means sufficiant food as your capacity but not over diagestion .only two time eating when u are strongly hunger & keep timing same for ever& maintain l am not taking any medicine nor allopethi or aurvedic .keep two times meal & get exercise daily for 4.5 km in 45 min.result will wt loss & diabetic reversion do it for one month & see result
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Thanks for your response.
Why I was asking you your daily calories intake and BMI because every person has got its own BMR (Basal Metabolic Rate).
You may be taking a HypoCalori Diet i.e your daily calories intake must be lower than your BMR. This perhaps has led to your weight loss and control in blood sugar.
It seems immaterial whether one takes two meals or 4 mini meals- both helps in curbing blood sugar provided that (1) one is on hypo calories and (2) on those food which does not cause a blood sugar spike i.e ultra Low GI foods.
It is great that many people have controlled blood sugar and reduced body weight by the above means. I had seen a post few days back where in UK the govt had supported a trial with a hypo calories Diet- 800 calories daily intake for controlling blood sugar and reducing body weight and the diet was a carb based diet. It is some kind of fasting to control blood sugar and achieving weight loss - it is no brainer ! If one is able to withstand such an ultra low calorie , it is great. But still it leaves a question mark on long term sustainability !
While it appears easier for high BMI T2 candidates to achieve both both Blood sugar control and weight loss simultaneously by following the well known methods as described above , it still remains a challenge for a low BMI 19-20 skinny guy like me to achieve Blood sugar control and at the same time gain some desirable weight.
Was seeking views from our active members if they have some inputs on the issue of blood sugar control and desirable weight gain for a Low BMI T2 guy or if some Low BMI T2 guy has done some thing and achieved this objective.
I do exercise almost 45 minutes 4.5KM. I will have my breakfast at 10 AM Luunch at 2 dinner at 8.30. No strict control on diet. At times I eat sweet. No sugar added to coffee or tea. My FBS is 160 PP is 220. I am diabetes since last 15 years and am 67 years old. HbA1c is 8.00 , i am under insulin and tablet morning 30 and night 30units. Very rarely my BG level goes to 75 random. What advice can you give to improve
Eating small meals every 4 hours also helps instead of going for two big meals and feel hungry most of the time . Lchf does n't make one to undergo hunger pangs , but long time practice makes one dissatisfied with that . with limit on portion size , healthy nutrient diet also keeps diabetes T2 under check with out any medicine what so ever . This is tested and proved . This is experimental research and observational evidence .
Not sure on Ayurveda but definitely can be reversed or put on remission like I did by changing the way u eat and activities. Good luck and u can do it.
Sorry.not possible.
'Cure' is a very relative term. Its meaning is different for different persons.
Everyone looking for cure, must first define what it means to them. Does it mean stopping medicines, or is it not having to care about food restrictions, or is it not having complications. Depending on that you will have to look for options.
One common meaning everyone has is 'no more medicines'.
And though it cant be possible for everybody, it is possible.
I don't know about Ayurveda. But i have seen it happen with homeopathy and some other diet therapies. As for Allopathy, it is not yet possible.
How is that possible? isnt it?
Let me first talk about Homeopathy. Homeopathic medicines for diabetes don't directly try to reduce sugar levels. Instead, they make the pancreas work better and increase its ability to break sugars normally.
If that happens, then automatically the need for taking medicines to control blood sugars on a daily level comes down.
And i have seen this happen. Patients are never advised to stop their allopathic anti-diabetic medicines when they start on Homeopathy. Because if they do that, it causes more problems.
So they have to continue their medicines and start Homeopathy side by side. The allopathic medicines will keep their daily sugars in control, while homeopathic medicines will stimulate the pancreas to improve its sugar control function.
This improvement can been seen on the FBS/PPBS in 3-6 months. Most importantly, there will be an improvement in the average sugar level which can be tested by measuring the HbA1c levels which will show the levels returning to near normal.
But all this is not the most important results. It is the improvement in the quality of life of the patients. Their ability to control their diet on their own and not out of compulsion or out of fear. Their mental attitude is much more positive and happy. Their improved ability to work and enjoy life.
For me, that is cure!
Again, as i said, there are some diet therapies also i came across while researching on the subject. You can check them out here: Freedom from Diabetes (youtube.com/user/FreedomFro... and Reversing Diabetes (sharan-india.org/diabetes-r...
If you know of some other approach, please let me know too.
This is what diabetes.co.uk has to say about use of homeopathy.
''Homeopathy
Homeopathy treatment is based on the use of highly diluted substances. Scientific research has shown homeopathic medicine to have no evidence of benefit beyond a placebo effect.
Homeopathy should not be taken to treat any health condition including any type of diabetes or diabetes related complications. It must not replace any prescription medication you are currently taking.''
At present, there is no cure for diabetes. However it can be controlled and in many instances reversed by modifications in diet and engaging in physical activities. And that is a lifestyle change. One can not go back to the old lifestyle.
It's not true that homeopathy cures diabetes. D is our inability to clear sugar from our blood. It's not a disease and pancreas is not at fault but our body cells esp muscles and liver are responding poorly to insulin.it's inability and can't be removed. Only if homeopathy can make us fly can it cure t2d.
Sorry, but the pancreas is certainly at fault. Beta cell dysfunction and insufficient beta cells are present in type 2 diabetics. This deteriorates over time.
ncbi.nlm.nih.gov/pmc/articl...
The page doesn't exist. There is a basic difference between 1 and 2. While 1 and lada etc are the disease of the pancreas, type 2 is not. In type 2, it's body cells that are diseased and not the pancreas. Pancreas is the victim. It's to overwork. This is what i believe.
How can one say that insufficient beta cells are present in type 2 diabetes? How is that found out in a living humanbeing? Why should beta cells die? What causes beta cells to die and why do those factors leave behind few/many cells? All the teaching and preaching in the past is proving wrong now.
There is one condition - the diabetes with two different pathology. That's what I believe.
The page does exist, and it is just one of the many scientific studies on the pancreas of type 2 diabetics. But, you are welcome to believe what you like.
It's not about individual belief. Whatever has been believed should have logic supported by evidence. We've to find out answers.
What do you mean, "supported by evidence"? I give you a working link and you say the page does not exist.
kapilv,
suramo is correct when saying that the link didn’t work. I have tried it myself twice and got the same message, unfortunately.
See my other reply. But the term is dysfunction! There's either to be deficiency in the insulin secretion or secretion of a faulty insulin. None of them is there. For type 2 Diabetes rather research should be targeted towards the cells not responding to insulin. All the research here to have been done on the theory that diabetes means fault of pancreas. That's why even today doctors do not advise or feel need to know the insulin levels of type 2 Diabetic people.
Can be really different in individual cases, sir. I speak from personal experience. When I had a fasting bg of 360+ and post-prandial of 500+,my fasted serum insulin was normal. I had it tested. My b.g was totally fine a few months ago, too.
So, it was rapidly escalating type 2 diabetes at age 36 for me. But, no hyperinsulinimia.
You may have crossed hyperinsulinimia stage leading to loss of beta cells and resulting in reduced insulin.
May be. But cell loss theory for t2d looked into with more scientific evidences. My question is what kills some cells and not all.
suramo
You are basically questioning LADA which is slow progression of beta cells destruction. There does not seem to be a clear cut answer to this in scientific literature.
Considering young age of kapilv there is a good possibility of LADA. We know that 10 % of T2D cases are misdiagnosed. They are in fact LADA.
There is no questioning on lada. Lada is an autoimmune disease. We're discussing here beta cell deaths in type 2 Diabetes. Irrespective of morphological changes in type 2 Diabetes, insulin secretion does occur. It's the beta cells that are not responding to that insulin. Morphological changes are the cause or result of overwork or some genetic defect(s ) need to ascertained.
You are right insulin secretion does occur in T2D, but I have seen many experimental results reported where insulin levels start dropping. That is when loss theory of beta cells come into picture. As you know, IR is compensated by beta cells in the beginning but later on it may start deteriorating in some cases and dropping to low level despite high BG. In fact, it is still a debate in scientific community about IR not being the sole cause of T2D.
If beta cells are kept in hostile environment they may die or get exhausted and give up.But what if that hostile atmosphere- high bs is removed? It's ir first. Beta cells are victims and not the cause.
Sounds convincing to me. Could you please give your comments on the statements/questions below:
1. The root cause of T2D is genetic. The diet( high carb) and inactive lifestyle merely accelerate the process to becoming diabetic and idm (low carb) + active lifestyle delay/reverse the conditions depending on when idm was implemented.
2. Is it true that first phase of insulin release capability is lost among all T2D?
3. Majority of obese people have IR but never become T2D.
Above is for my learning. I know you are an expert.
Valid questions. Debated here many times.
1) my understanding and personal experience is different. The disease process starts early in the life. There is no overt diabetes but the inability to consume carbs starts since then. Obviously we feel lousy. We don't do / feel good at exercising. So it's the result. My understanding needs more research. It can be both ways but only exercise or active life may not avoid D.I had always been very active, playing cricket when diagnosed D 14 years before.
2) yes.It's postulated. If there is a research article on it share it / them here.It's also postulated that pancreas empties the entire insulin stock once we start eating and then for 55 min there is no insulin secretion even if we eat carby food.I don't buy this theory but i have a knowledge that Insulin starts secreting once bs cross 100 mg / ml level and drops once bs comes down below that.
3) yes.True.That's true for non obese people too.Also the sugar processing capacity differs even among diabetics. Some D people eat carbs but their a1c / bs remain reasonably under control and do not develop severe complications. Not true for very high carb dietars or for those who are eating irrationally. On the contrary large % of D people never care for what to eat.They eat like normal nonD people and live a complication free lives.
We still need to learn much more. Why insulin level down and a1c high as against insulin high with excellent a1c.
What're your insulin levels? yes.Different in different people. Every t2d has different capacity to clear glucose. In my case too insulin levels very well low ( 3.31 fasting & 10.2 pp) but a1c yet not under control. It's bounced. I've observed that less carbs i take higher is the fbs. We need to learn a lot. May be those who have excellent bs control and a1c @5 may have higher insulin levels than mine.
We need to look at this with scientific evidences.The people must be tested / screened repeatedly to ensure there is beta cell loss.Beta cells do not replicate or their postulated loss could be compensated by new cells.There's a possibility that genetic defects causing body cells to become insulin resistant also have effects on beta cells but still there is enough or more insulin. And if it's beta cell dysfunction how can people reverse their diabetes and remain euglycemic for their whole life with idm?
Let me add further that we as diabetics are not interested in academic discussions. More than number and morphology of beta cells i'm interested in their functioning. All such studies have been undertaken with a biassed belief that pancreas is at fault. The dysfunction can be attributed to beta cells only if they fail to secrete enough insulin as in t1 and lada or secretes defective insulin that fails to act on body cells while the body cells is responding to the right insulin. If that's the case then transplanting right insulin producing beta cells will cure t2d.But that's not the case. So for t2d i'd not use beta cell dysfunction.
Further glucose and lipid toxicities said to be the cause of some beta cells !! So why should only some beta cells die and not all?
Loads of journals on it.
care.diabetesjournals.org/c...
Although it has long been assumed that insulin resistance is the leading factor in the pathogenesis of type 2 diabetes (1), evidence for the importance of the pancreatic β-cells has accumulated over the past decades. In fact, the vast majority of genes associated with type 2 diabetes have been linked to the β-cell, and impairments in β-cell mass and in insulin secretion have been reported in numerous studies in patients with type 2 diabetes. One misconception that has prevented the appreciation of the β-cell defects for a long time is the idea of a “hyperinsulinemia” in patients with type 2 diabetes. This concept has arisen from the observation that patients with type 2 diabetes often present with higher fasting insulin concentrations than nondiabetic individuals. However, if insulin concentrations are interpreted in the context of the concurrently elevated glucose levels in patients with type 2 diabetes, a relative insulin deficit rather than hyperinsulinemia becomes apparent. Furthermore, when insulin secretion is evaluated under stimulated conditions (e.g., after intravenous glucose administration), the typical defects, especially in early-phase insulin release, can be unmasked (2,3).
Is type 2 diabetes primarily caused by a deficit in β-cell mass?
That type 2 diabetes develops largely because of a deficit in β-cell mass is supported by several lines of evidence. Autopsy studies in various populations (European, Asian, and North American) have reported significant reductions in the amount of pancreatic β-cells in patients with type 2 diabetes compared with nondiabetic individuals (5–7). The extent of this deficit ranges from ∼20% in some studies to ∼65% in others (5–7). There is also evidence for a β-cell deficit in prediabetic individuals with impaired fasting glucose (6).