Robert Rogers, PhD: 2018 update

Robert Rogers, PhD - 2018 Update of Road to Recovery from Parkinsons Disease

parkinsonsdisease.me/

The 2018 Update covers the following:

• High Dose Thiamine; blog.parkinsonsrecovery.com...

•Pulsed Electromagnetic Field Therapy (PEMF)

•Sleep Therapies

•Ketone Esters

•CBD Oils

•Macrobiotics

•Amino Acid Therapy

[parkie13]

Something interesting in the article, the last sentence Robert Rogers article is once the methylation cycle is established you don't require any more high doses of B1. Does it mean don't require it for a while, does its mean you have to start it again when the process stops, it has not been explained very well.

[Hidden in reply to parkie13]

Maybe the cycle is not ever reestablished without continuously using B1 for those PwP.

[Kia17 in reply to parkie13]

parkie13

I think it means after methylation cycle fixed you are fully recovered from the PD.

Kia

[Erniediaz1018]

Great share Roy, thanks.

[Hidden]

Well he presents a different theory than Dr. Costantini on why B-1 might be helpful in PD, but I don't follow his thinking. He blames a short circuited methylation cycle that does not allow proper utilization of the B vitamins by the body, but if it is really "short circuited" as he describes in the article, it is not working and more B-1 would not make a difference under those conditions! Lacking studies to prove what and why B-1 does what it does in PD, I think Dr. Costantini's theory is more plausible! He leans toward a defective or diminished capacity transporter mechanism for thiamine to the brain in which case more thiamine intake would equate with more thiamine to the brain. This could easily explain for the wide range of effective dosing that we have seen on this forum!

Art

[Niggs in reply to Hidden]

I agree Art, and also why benefit is found in PwP's taking B1 regardless of their thiamine levels. Would one not also expect to possibly see signs of thiamine deficiency if the body is unable to utilize B1 properly ? I'm treading carefully as I have little knowledge on this particular area and have to state B1 therapy is new to me, I intend to trial it once my prescribed med adjustments have settled (if they ever do) but just prior to this I dabbled a bit. Nothing concrete to report but enough to convince me B1 'does' effect dopamine levels and thus pd. My first observation is that it may behave similar to an agonist. I've been on Requip xl for 9yrs and changed doses more times than you can shake a stick at ! There are certain biomarkers I know are due to when the agonist dosage is changed, increased libido, urinary frequency, temporary mild dyskinesia etc. I experienced these same biomarkers on introducing B1.

I'll write a full report I've trialled it properly.

Nigel

[Hidden in reply to Niggs]

Reply to Niggs,

That would be helpful if it acts as an agonist, because according to park_bear, standard med agonists may be a problem for some PWPs! If you are correct, then thiamine may offer another option to the dopamine agonists, but a study to prove your theory seems fairly unlikely, so here we are once again left to our own devices to try and figure out what may help!

Art

[Niggs in reply to Hidden]

I may be wrong but I seem to recall Dr C, in a taped Q&A mentioning that the DA element may need reducing with his thiamine protocol in some folk, just another possible clue. Yes I would say there's not a snowball's chance in hell of any study being done Art,

'Patient heal thyself !'