There’s still a lot of confusion across the nation about whether or not marijuana is effective for cancer patients. Odds are you’ve heard something about it but weren’t sure whether the information was reliable or definitive. So, in order to help clear things up, here is a list of 34 studies showing that marijuana cures cancer, categorized by the type of cancers being cured in each study. As you sort through the articles, note that the consistent theme between them is that cannabis shrinks tumors and selectively targets cancer cells. As bills and voter initiatives to legalize medical marijuana spread from state to state, remember that we’re not just talking about mitigating the side effects of chemo (though this is another viable use), we’re talking about curing the cancer itself as well as preventing its spread. I’ve taken the liberty of only including articles from credible scientific journals, removing any biased or otherwise improperly cited studies. Enjoy!
Cures Brain Cancer
Cures Mouth and Throat Cancer
Cures Breast Cancer
Cures Lung Cancer
Cures Uterine, Testicular, and Pancreatic Cancers
Cures Prostate Cancer
Cures Colorectal Cancer
Cures Ovarian Cancer
Curse Blood Cancer
Cures Skin Cancer
Cures Liver Cancer
Cures Biliary Tract Cancer
Cures Bladder Cancer
medscape.com/viewarticle/80... (Sign-up required to view study)
Cures Cancer in General
Sent from the Borge mother ship.
On 17 Oct 2013, at 07:07, Stevehemp <firstname.lastname@example.org> wrote:
I know it work it worked for my ,, I would really like somebody to look at my case file ,, it's important not so Much for me as I am cured , my live is fine now ,my dr is stunned ,, but can not use the info , I been told by my hepc dr it is a miracle cure ,,,
,, cannibis cures ,it's a fact ,,
I know it not very P.c , but the fact remains that people are dropping dead and for us hep.c liver cancer , it not a pleasant death ,I know I nearly went twice when I did the in ter fear on , that treatment is evil and now unnecssersy, try , thc oil first , if does not work ,no harm done , and if it does work , no nasty side affects ,bit light headed and laugh a lot , vs in ter fear on , well don't go there ,, that a trip to hell , that to many don't come back from , and if you are Lucky - a year on treatment , and the recovery after ,, that's a long haul back , you know you been there to ,, please put this on file please try and get it out there , there is over 1600 per reviews on cannibis from the who's who's ,of research centres, the a list of university's for a round the world , the evidence is totally overwhelming , in favour of cannibis , check it out , google ? Ted talks , charlottes web , watch it make you own mind up , check my medical recorders , France legal ,Spain. ,USA 26 states legal ,,
If nothing more than use the info for your self or give it to somebody who might use the info xx
Anti-tumoral action of cannabinoids on hepatocellular carcinoma: role of AMPK-dependent activation of autophagy.
AuthorsVara D, et al. Show allJournal
Cell Death Differ. 2011 Jul;18(7):1099-111. doi: 10.1038/cdd.2011.32. Epub 2011 Apr 8.
Department of Biochemistry and Molecular Biology, School of Medicine, Alcalá University, Madrid, Spain.
Cell Death Differ. 2011 Jul;18(7):1237.
Hepatocellular carcinoma (HCC) is the third cause of cancer-related death worldwide. When these tumors are in advanced stages, few therapeutic options are available. Therefore, it is essential to search for new treatments to fight this disease. In this study, we investigated the effects of cannabinoids--a novel family of potential anticancer agents--on the growth of HCC. We found that Δ(9)-tetrahydrocannabinol (Δ(9)-THC, the main active component of Cannabis sativa) and JWH-015 (a cannabinoid receptor 2 (CB(2)) cannabinoid receptor-selective agonist) reduced the viability of the human HCC cell lines HepG2 (human hepatocellular liver carcinoma cell line) and HuH-7 (hepatocellular carcinoma cells), an effect that relied on the stimulation of CB(2) receptor. We also found that Δ(9)-THC- and JWH-015-induced autophagy relies on tribbles homolog 3 (TRB3) upregulation, and subsequent inhibition of the serine-threonine kinase Akt/mammalian target of rapamycin C1 axis and adenosine monophosphate-activated kinase (AMPK) stimulation. Pharmacological and genetic inhibition of AMPK upstream kinases supported that calmodulin-activated kinase kinase β was responsible for cannabinoid-induced AMPK activation and autophagy. In vivo studies revealed that Δ(9)-THC and JWH-015 reduced the growth of HCC subcutaneous xenografts, an effect that was not evident when autophagy was genetically of pharmacologically inhibited in those tumors. Moreover, cannabinoids were also able to inhibit tumor growth and ascites in an orthotopic model of HCC xenograft. Our findings may contribute to the design of new therapeutic strategies for the management of HCC.
PMID 21475304 [PubMed - indexed for MEDLINE]
PMCID PMC3131949 Free Full Text
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