My scans showed no spread but Sept 1 my right nipple became very sensitive and a 2" diameter hardness formed around it. I applied Clio twice a day and today the hardened area is the size of a dime. The Clio I use is 5-chloro-7-iodoquinolin-8-ol). U of Maryland is the best source of cancer research that I have found. They report on prolactin dependent prostate cancer which has no markers like a PSA and more die from it than testosterone dependent prostate cancer. Check them out.
My scans showed spread but...... - Advanced Prostate...
My scans showed spread but......
Suggestion how to access that UM research?? Also, where did you find that info about prolactin dependent prostate cancer , and its lethality compared to testosterone dependent PCa?
Mathews Journal search "Clioquinol". Two cases of prostate cancer terminated.
The cabergoline treatment of the patient would terminate that malignancy if it were present. Otherwise, the patient unknowingly would have died of prolactin-dependent prostate cancer. That is the reason for my reference of prolactin-dependent prostate cancer as the “silent killer” that is responsible for most of the deaths due to prostate cancer.
ABSTRACT
All cases of prostate cancer exhibit the hallmark condition of marked decrease in zinc in malignancy compared to the high zinc levels in the normal and benign prostate. There exists no reported corroborated case of prostate cancer in which malignancy exhibits the high zinc levels that exist in the normal prostate acinar epithelium. The decrease in zinc is achieved by the downregulation of ZIP1 zinc transporter, which prevents the uptake and accumulation of cytotoxic zinc levels. Thus, prostate cancer is a “ZIP1-deficient” malignancy. Testosterone and prolactin are the major hormones that similarly regulate the growth, proliferation, metabolism, and functional activities of the acinar epithelial cells in the peripheral zone (the site of development and progression of malignancy). Testosterone regulation provides the basis for androgen ablation treatment of advanced prostate cancer, which leads to the development of terminal androgen-independent malignancy. Androgen- independent malignancy progresses under the influence of prolactin. These relationships provide the basis for the prevention and treatment of advanced prostate cancer. Clioquinol (zinc ionophore; 5-chloro-7-iodoquinolin-8-ol) is employed to facilitate zinc transport and accumulation in the ZIP1-deficient malignant cells and induce cytotoxic effects. Cabergoline (dopamine agonist) is employed to decrease prolactin production and its role in the progression of androgen-independent malignancy. We propose a clioquinol/cabergoline treatment regimen that will be efficacious for aborting terminal advanced prostate cancer. FDA policies permit this treatment regimen to be employed for these patients.
Key words: Androgen-independent prostate cancer, cabergoline bromocriptine agonist, clioquinol zinc ionophore, testosterone and prolactin
It's very surprising that I've never heard of this nor seen anyone refer to this treatment given that they say prolactin-dependent prostate cancer "is responsible for most of the deaths due to prostate cancer". After reading the Mathews Journal article, I wonder why it isn't a mainstream SOC treatment and would be interested to see what others say about it.
BTW this is the article : mathewsopenaccess.com/full-...
Those two cases of terminated prostate cancer is why I began the protocol on Aud 11. I believe it's working for me. Because it's too expensive to test for prolactin cancer I'm just assuming I have and take prolactin reducing meds. The reason its not mainstream is money.
Two ounces of clioquinol was $125 and will last about two months.