I started my resistant starches today. Today it was a green banana in my smoothie. Tomorrow my bag of resistant starch (type 2, green banana powder) arrives.
They used RS-3 in the study and I can't find any RS-3 supplements. This is odd because they specifically point out that they used a convenient supplement that was mixed with water. This is what I found for RS-3: Cooked and cooled potato, bread and cornflakes.
They did reference another study with even better results using RS-2, so that will have to do for now. I may add in some cooked and cooled potatoes.
Reasons for adding resistant starches:
1 - anti-inflammatory effects indicated by the decline in fecal calprotectin concentrations.
2 - increase in butyrate concentrations.
3 - a significant improvement with regard to depressive symptoms.
"Resistant starch type 2 (RS2). This type of starch is naturally resistant because of the nature of the starch granule. RS2 occurs in foods where the starch is eaten raw (e.g. unripe bananas)"
Don't cook the potato. peel it, cut it in pieces and refrigerate it. then salt it and eat it raw.
I love them raw. Use Idaho potatoes. try a number as every potato doesnt taste good.
I have been using potato starch for about 10 weeks now. One ten week study reported an improvement in constipation by the end. My GI system has improved a bit but it may be because I also added prune juice. I suppose I should experiment with just one substance at a time,
"In contrast to our initial hypothesis, symptoms related to constipation (a frequent non-motor symptom in PD) were not significantly altered during the 8-week-intervention."
Primary outcome measures were: change (prior vs. post intervention) in a) bowel habits....
which raises several questions they have trouble answering:
"While the study by Alfa and colleagues [19] even indicated a therapeutic effect (reduction in the use of laxatives), we did not observe a significant improvement of bowel habits. This divergent observation between our study and the study by Alfa and colleagues might be due to different types of RS (we used RS type 3, Alfa and colleagues used RS type 2), the dose of RS (Alfa and colleagues administered approximately double the dosage compared to our study), and the duration of the interventional period (8 weeks in our study versus 12 weeks in the study by Alfa and colleagues)."
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