I finally found a study livingwithatrialfibrillatio... spinal issues and AF.
This website gives a layperson's brief summary:
clarkchiropractic.net/irreg...
And you may find this site interesting:
livingwithatrialfibrillatio...
A bit of the discussion in the study:
The present study reveals that CS is associated with a 3.10-fold increase of arrhythmia risk compared to patients without cervical spondylosis, especially atrial fibrillation, ventricular and supraventricular tachycardia. ANS stimulation could explain the related mechanism of this finding. Clinical evidence had shown that CS can cause sympathetic nerve irritation and associated sympathetic symptoms [27], and instability at the C4–C5 intervertebral space is the most-common type causing sympathetic symptoms [28]. Recognized sympathetic symptoms of CS are vertigo, dizziness, tinnitus, headache, and palpitation [29]. Previous electrophysiological studies have demonstrated that sympathetic activation has a proarrhythmic effect in enhancing automaticity, triggering re-entry, and reducing the threshold of refractoriness [29,30,31]. Such studies have shown that an imbalance of the autonomic nervous system and stimulation of the sympathetic nervous system can trigger atrial fibrillation [32], ventricular tachycardia [33], and ventricular fibrillation [34]. Such findings are thus similar to those of our study: CS is associated with a higher risk of developing atrial fibrillation, atrial flutter, and ventricular tachycardia. We supposed that a possible mechanism would be that cervical region trauma influences spinal sympathetic neurons because sympathetic control of heart originates from T1-5, thus the parasympathetic tone is unopposed. The increased vagal tone would be associated with atrial fibrillation. A reason for the insignificant risk of developing ventricular fibrillation shown in our study is possibly related to low event rate which only one event occurred in the study cohort and comparison cohort individually.