I have read several papers indicating a connection between the process referred to as "methylation." It's complicated, as is everything connected with prostate cancer and, to be truthful, I find it very difficult to follow the discussions in these papers. Therefore, I am putting this communication out there for commentary from those of you who understand methylation. It appears, that cancer sells may grow when it is too prevalent by utilizing, for their benefit, the glutathione created during Methylation. Glutathione normally protects cells from ROS. Why am I concerned? If this is true, cancer patients need to pay attention to whatever contributes to methylation or increases their glutathione levels. For example, I have stopped taking folate and N-Acetyl-L-Cysteine. I was sorry to give up the NAC because it, among other things, is a good detoxifier of the liver and, considering all of the toxins (read: medications) that I am exposed to on a daily basis, this is a big drawback. Can anyone clarify for me how to deal with methylation (we need it, but not too much!), so that I may, through diet, benefit from it, yet not suffer any adverse effects. Thanks.