Six week-ago I got my Pacemaker, due to a very slow heart rate (bradycardia) for three days after the implant of Pacemaker I did not take any Bisoprolol. I felt quite good and my heart rate was settling down at 60. At the fourth day follow-up visit after implantation, Dr. ordered Bisoprolol increase from 1.25mg to 2.5 mg. I had constant episods of arrhytmia, much worst than before the Pacemaker. I had cut back on the Bisoprolol to the privous dose of 1.25 mg and gradually to .625mg and to zero. I felt that the Bisoprolol was trying to push my heart rate below the Pacemaker setting of 60. Now I am thinking that my original slow rate problem was caused by Bisoprolol to begin with.
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I am on the lowest dose of Diltiazem which is prescribed if you can't take betablockers. I am not allowed to take a higher dose because when I was taking it my heart paused for a few seconds occasionally. At the moment we are waiting to see how things go but if I have frequent fast AF I will have to have a pacemaker so I can take the higher dose.
I have no idea if your theory is correct but wonder if the Bisoprolol was also for high blood pressure which my medication is, as then it should be replaced with something for high BP?
I'm not a medic but, as I understand it, the objective of beta blockers (or calcium channel blockers like Diltiazem) is to reduce the peak heart rate. Reductions in the resting heart rate are, as it were, a side effect.
In my case, I needed a pacemaker to stop my heart from pausing (and causing me to black out) not as a result of too low a resting heart rate.
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