Alzheimer's is experiencing a paradigm shift. Nonetheless, publicity about the important findings is seemingly nonexistent. This is somewhat akin to media silence about Costantini and HDT.
Here are Important findings about herpes, Alzheimer's, and dementia. Amyloid beta and Tau are antimicrobial peptides. Oodles of suppoting data. Quite exciting!
Anti-herpetic Medications and Reduced Risk of Dementia in Patients with Herpes Simplex Virus Infections—a Nationwide, Population-Based Cohort Study in Taiwan : 2018.
Chronically treating symptoms has long been profitable. Thus it came to pass that avoiding cures became an operational method for medicationists and their Big Pharma overlords.
For your reading pleasure. I am 99% sure that when immuno therapy study is done 5 years and a billion dollars later we're going to be informed that alpha syN have secondary role in pd.
So do you think it is viruses and toxins doing the damage and the alpha syn is just the bodies way of entrapping the viruses? Perhaps the blobs of protein cause some of the symptoms by gumming up things but prevent the toxins doing more damage? So dissolving the protein clumps might rerelease the viruses and toxins into the body?
Lewy body diseases are characterized by the presence of Lewy bodies, alpha-synuclein(AS)-positive inclusions in the brain. Since their main component is conformationally modified AS, aggregation of the latter is thought to be a key pathogenic event in these diseases.
The analysis of inclusion body constituents gives additional information about pathways also involved in the pathology of synucleinopathies.
Widespread mitochondrial dysfunction is very closely related to disease development. The impairment of protein degradation pathways, including both the ubiquitin-proteasome system and the autophagy-lysosome pathway also play an important role during the development of Lewy body diseases.
Finally, differential expression changes of isoforms corresponding to genes primarily involved in Lewy body formation point to alternative splicing as another important mechanism in the development of Parkinson’s disease, as well as dementia with Lewy bodies.
The present paper attempts to give an overview of recent molecular findings related to the pathogenesis of Lewy body diseases.
Molecular Pathology of Lewy Body Diseases.
Katrin Beyer, Montserrat Domingo-Sàbat, and Aurelio Ariza.
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