Here is a miscellany of references relevant to Sulzer et al 2017,
who did not claim that autoimmunity is the only causal factor in PD.
Indeed, anti-alpha-synuclein may be an etiologically significant
co-factor in only a subset of PwP.
All but one is open access. Be forewarned, mmunology has its own
Inflammation and Adaptive Immunity in PD.
The Role of Innate and Adaptive Immunity in PD.
MHC-I expression renders catecholaminergic neurons susceptible to
PD-associated genetic variation is linked to quantitative expression
of inflammatory genes.
Common Genetic Variant Association with Altered HLA Expression,
Synergy with Pyrethroid Exposure, and Risk for PD: An Observational
and Case-Control Study.
Age and duration of inflammatory environment differentially affect
the neuroimmune response and catecholaminergic neurons in the
midbrain and brainstem.
Role of cytokines in inflammatory process in Parkinson's disease.
Distribution of major histocompatibility complex class II-positive
microglia and cytokine profile of Parkinson's disease brains.
Association of Parkinson disease with structural and regulatory
variants in the HLA region.